Search results for "Schwann Cells"

showing 10 items of 18 documents

Functions of histone modifications and histone modifiers in Schwann cells.

2019

Schwann cells (SCs) are the main glial cells present in the peripheral nervous system (PNS). Their primary functions are to insulate peripheral axons to protect them from the environment and to enable fast conduction of electric signals along big caliber axons by enwrapping them in a thick myelin sheath rich in lipids. In addition, SCs have the peculiar ability to foster axonal regrowth after a lesion by demyelinating and converting into repair cells that secrete neurotrophic factors and guide axons back to their former target to finally remyelinate regenerated axons. The different steps of SC development and their role in the maintenance of PNS integrity and regeneration after lesion are c…

0301 basic medicine570 Life sciencesLesionHistones03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineNeurotrophic factorsPeripheral Nerve InjuriesmedicineAnimalsHumansSecretionTranscription factorMyelin SheathbiologyRegeneration (biology)AxonsCell biologyNerve Regeneration030104 developmental biologyHistonemedicine.anatomical_structurenervous systemNeurologyMyelin sheathPeripheral nervous systembiology.proteinSchwann Cellsmedicine.symptom030217 neurology & neurosurgery570 BiowissenschaftenGliaREFERENCES
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Neurofibromatosis type 2 tumor suppressor protein is expressed in oligodendrocytes and regulates cell proliferation and process formation.

2017

The neurofibromatosis type 2 (NF2) tumor suppressor protein Merlin functions as a negative regulator of cell growth and actin dynamics in different cell types amongst which Schwann cells have been extensively studied. In contrast, the presence and the role of Merlin in oligodendrocytes, the myelin forming cells within the CNS, have not been elucidated. In this work, we demonstrate that Merlin immunoreactivity was broadly distributed in the white matter throughout the central nervous system. Following Merlin expression during development in the cerebellum, Merlin could be detected in the cerebellar white matter tract at early postnatal stages as shown by its co-localization with Olig2-positi…

0301 basic medicineCentral Nervous SystemCytoplasmlcsh:MedicineNervous SystemMyelinMiceCell MovementAnimal CellsCerebellumMedicine and Health SciencesNeurofibromatosis type 2lcsh:ScienceNeuronsStainingCerebral CortexNeurofibromin 2MultidisciplinarybiologyCell StainingBrainCell migrationCell biologyOligodendrogliamedicine.anatomical_structureGenetic DiseasesCell ProcessesAnatomyCellular TypesCellular Structures and OrganellesResearch ArticleCell typeNeurofibromatosis 2NeurogenesisNerve Tissue ProteinsTransfectionResearch and Analysis MethodsCell Line03 medical and health sciencesmedicineAnimalsImmunohistochemistry TechniquesCell ProliferationCell NucleusClinical GeneticsCell growthAutosomal Dominant Diseaseslcsh:RBiology and Life SciencesCell Biologymedicine.diseaseOligodendrocyteMyelin basic proteinMerlin (protein)Mice Inbred C57BLHistochemistry and Cytochemistry Techniques030104 developmental biologySpecimen Preparation and TreatmentAstrocytesNeurofibromatosis Type 2Cellular Neurosciencebiology.proteinImmunologic Techniqueslcsh:QSchwann CellsNeurosciencePLoS ONE
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Irreversible changes occurring in long-term denervated Schwann cells affect delayed nerve repair.

2017

OBJECTIVEMultiple factors may affect functional recovery after peripheral nerve injury, among them the lesion site and the interval between the injury and the surgical repair. When the nerve segment distal to the lesion site undergoes chronic degeneration, the ensuing regeneration (when allowed) is often poor. The aims of the current study were as follows: 1) to examine the expression changes of the neuregulin 1/ErbB system during long-term nerve degeneration; and 2) to investigate whether a chronically denervated distal nerve stump can sustain nerve regeneration of freshly axotomized axons.METHODSThis study used a rat surgical model of delayed nerve repair consisting of a cross suture betw…

0301 basic medicinePathologymedicine.medical_specialtyTime FactorsNerve rootNeuregulin-1Settore MED/19 - Chirurgia PlasticaSchwann cellNRG1/ErbB system03 medical and health sciences0302 clinical medicinePeripheral Nerve InjuriesMedicineAnimalsNeuregulin 1Rats Wistardelayed nerve repairDenervationneuregulin 1biologybusiness.industryRegeneration (biology)General MedicineAnatomyRecovery of FunctionDenervationMedian nerveNerve RegenerationRats030104 developmental biologymedicine.anatomical_structureperipheral nervePeripheral nerve injuryNerve Degenerationstereologybiology.proteinFemaleSchwann CellsbusinessEpineurial repair030217 neurology & neurosurgeryJournal of neurosurgery
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Sox8 and Sox10 jointly maintain myelin gene expression in oligodendrocytes

2017

In Schwann cells of the vertebrate peripheral nervous system, induction of myelination and myelin maintenance both depend on the HMG-domain-containing transcription factor Sox10. In oligodendrocytes of the central nervous system, Sox10 is also essential for the induction of myelination. Its role in late phases of myelination and myelin maintenance has not been studied so far. Here, we show that these processes are largely unaffected in mice that lack Sox10 in mature oligodendrocytes. As Sox10 is co-expressed with the related Sox8, we also analyzed oligodendrocytes and myelination in Sox8-deficient mice. Again, we could not detect any major abnormalities. Expression of many myelin genes was …

0301 basic medicineSOX10Central nervous systemGene ExpressionBiologyMice03 medical and health sciencesCellular and Molecular NeuroscienceMyelin0302 clinical medicineGene expressionmedicineAnimalsHumansCell LineageGeneMyelin SheathMice KnockoutSOXE Transcription FactorsHEK 293 cellsOligodendrocyteOligodendrogliaHEK293 Cells030104 developmental biologymedicine.anatomical_structurenervous systemNeurologyMyelin maintenanceembryonic structuresSchwann CellsNeuroscience030217 neurology & neurosurgeryHeLa CellsGlia
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A predominantly glial origin of axonal ribosomes after nerve injury

2017

Axonal mRNA transport and local protein synthesis are crucial for peripheral axon regeneration. To date, it remains unclear how ribosomes localize to axons. They may be co-transported with mRNAs or, as suggested by recent studies, transferred from Schwann cells (SC). Here, we generated transgenic "RiboTracker" mice expressing tdTomato-tagged ribosomal protein L4 in specific cell types when crossed with Cre lines. Two neuronal RiboTracker-Cre lines displayed extremely low levels of axonal L4-tdTomato-positive ribosomes. In contrast, two glial RiboTracker-Cre lines revealed tagged ribosomes in sciatic nerve (SN) axons with increasing amounts after injury. Furthermore, non-RiboTracker dorsal r…

0301 basic medicineSchwann cellMice TransgenicBiologyRibosome03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicinePeripheral Nerve InjuriesRibosomal proteinGanglia SpinalmedicineProtein biosynthesisAnimalsMRNA transportAxonNerve injurySciatic NerveAxonsNerve RegenerationCell biology030104 developmental biologymedicine.anatomical_structurenervous systemNeurologySchwann CellsSciatic nervemedicine.symptomNeuroglia030217 neurology & neurosurgeryGlia
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11q Deletion or ALK Activity Curbs DLG2 Expression to Maintain an Undifferentiated State in Neuroblastoma

2020

High-risk 11q deleted neuroblastomas typically display undifferentiated/poorly differentiated morphology. Neuroblastoma is thought to develop from Schwann cell precursors and undifferentiated neural crest (NC) derived cells. It is therefore vital to understand mechanisms involved in the block of differentiation. We identify an important role for oncogenic ALK-ERK1/2-SP1 signaling in maintenance of undifferentiated NC-derived progenitors via repression of DLG2, a tumor suppressor in neuroblastoma. DLG2 is expressed in the ‘bridge signature’ that represents the transcriptional transition state when neural crest cells or Schwann Cell Precursors become chromaffin cells of the adrenal gland. We …

0301 basic medicineTranscription GeneticCarcinogenesisChromaffin CellsRetinoic acidlaw.inventionNeuroblastomachemistry.chemical_compound0302 clinical medicinelawNerve Growth FactorMedicine and Health Sciencesretinoic acidAnaplastic Lymphoma Kinaselcsh:QH301-705.5NeuronsMice Inbred BALB CNeural crestCell DifferentiationPrognosisCandidate Tumor Suppressor GeneDLG2Up-RegulationCell biologyGene Expression Regulation NeoplasticERKPhenotypeTreatment Outcomemedicine.anatomical_structureFemaleChromosome Deletiontumor suppressorMAP Kinase Signaling SystemSp1 Transcription FactorSchwann cellGenetics and Molecular BiologyTretinoinBiologyGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesAdrenergic AgentsCell Line TumorNeuroblastomamedicineAnimalsHumansProgenitor cellGenePsychological repressionCell ProliferationChromosomes Human Pair 11Tumor Suppressor Proteinsmedicine.disease030104 developmental biologyALKlcsh:Biology (General)chemistryTrk receptorGeneral BiochemistrySuppressorSchwann CellsGuanylate Kinases030217 neurology & neurosurgerySSRN Electronic Journal
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Injured Axons Instruct Schwann Cells to Build Constricting Actin Spheres to Accelerate Axonal Disintegration

2019

Summary: After a peripheral nerve lesion, distal ends of injured axons disintegrate into small fragments that are subsequently cleared by Schwann cells and later by macrophages. Axonal debris clearing is an early step of the repair process that facilitates regeneration. We show here that Schwann cells promote distal cut axon disintegration for timely clearing. By combining cell-based and in vivo models of nerve lesion with mouse genetics, we show that this mechanism is induced by distal cut axons, which signal to Schwann cells through PlGF mediating the activation and upregulation of VEGFR1 in Schwann cells. In turn, VEGFR1 activates Pak1, leading to the formation of constricting actomyosin…

Male0301 basic medicineCellGeneral Biochemistry Genetics and Molecular BiologyCell LineMice03 medical and health sciences0302 clinical medicinePAK1Downregulation and upregulationPeripheral Nerve InjuriesmedicineAnimalsRats WistarAxonlcsh:QH301-705.5Cells CulturedActinPlacenta Growth FactorVascular Endothelial Growth Factor Receptor-1ChemistryActinsAxonsRatsCell biologyMice Inbred C57BLOligodendrogliaCrosstalk (biology)030104 developmental biologymedicine.anatomical_structurelcsh:Biology (General)p21-Activated Kinasesnervous systemcardiovascular systemNerve lesionFemaleSchwann Cells030217 neurology & neurosurgerySignal TransductionClearanceCell Reports
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Degradable poly(amidoamine) hydrogels as scaffolds for in vitro culturing of peripheral nervous system cells.

2012

This paper reports on the synthesis and physico-chemical, mechanical, and biological characterization of two sets of poly(amidoamine) (PAA) hydrogels with potential as scaffolds for in vivo peripheral nerve regeneration. They are obtained by polyaddition of piperazine with N,N′-methylenebis(acrylamide) or 1,4-bis(acryloyl)piperazine with 1,2-diaminoethane as cross-linking agent and exhibit a combination of relevant properties, such as mechanical strength, biocompatibility, biodegradability, ability to induce adhesion and proliferation of Schwann cells (SCs) preserving their viability. Moreover, the most promising hydrogels, that is those deriving from 1,4-bis(acryloyl)piperazine, allow the …

Materials Chemistry2506 Metals and AlloysPoly(amidoamine)Cell SurvivalBioengineeringBiocompatible MaterialsNeural cell culturingPiperazinesRats Sprague-DawleyGanglia SpinalCell AdhesionPolyaminesAnimalsCell ProliferationNeuronsAcrylamidesPolymers and PlasticTissue EngineeringTissue ScaffoldsHydrogelsPolymer applicationEthylenediaminesBiomaterialNerve RegenerationRatsHydrogelBiodegradableSchwann CellsBiotechnologyMacromolecular bioscience
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The fine structure of de-and reinnervated muscle spindles

1974

Reinnervated muscle spindles in lower lumbrical muscles of rats studied 17 days to 24 months after crushing the sciatic nerve showed a series of alterations which have not been analysed, thus far, by electron microscopy. There was a striking increase of the number of intrafusal muscle fibers seen in approximately 20% of reinnervated spindles. These spindles showed 5–11 intrafusal muscle fibers whereas normal spindles usually contained 3–4 fibers only.

Nerve CrushMuscle spindleMotor nerveSensory systemBasement MembranePathology and Forensic Medicinelaw.inventionCellular and Molecular NeuroscienceMuscle pathologyMyofibrilslawmedicineAnimalsMuscle SpindlesMotor NeuronsMuscle DenervationChemistryAnatomySciatic NerveAxonsMuscle DenervationNerve RegenerationRatsMicroscopy ElectronIntercellular Junctionsmedicine.anatomical_structureFemaleSchwann CellsNeurology (clinical)Sciatic nerveElectron microscopeWallerian DegenerationActa Neuropathologica
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Wrapping it up: the cell biology of myelination.

2007

During nervous system development, oligodendroglia in the central nervous system (CNS) and Schwann cells in the peripheral nervous system (PNS) synthesise large amounts of specific proteins and lipids to generate myelin, a specialised membrane that spirally ensheathes axons and facilitates fast conduction of the action potential. Myelination is initiated after glial processes have attached to the axon and polarisation of the plasma membrane has been triggered. Myelin assembly is a multi-step process that occurs in spatially distinct regions of the cell. We propose that assembly of myelin proteins and lipids starts during their transport through the biosynthetic pathway and continues at the …

Nervous systemProteolipid protein 1General NeuroscienceCentral nervous systemMyelin Basic ProteinBiologyNeuroregenerationModels BiologicalMyelin assemblyCell biologyMyelinmedicine.anatomical_structurenervous systemPeripheral nervous systemmedicineAnimalsSchwann CellsAxonNeuroscienceBiologyMyelin ProteinsCurrent opinion in neurobiology
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